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Mongi Ben Hamida
Mongi Ben Hamida (Arabic: منجي بن حميدة), born 1 February 1928 in Kelibia, and died on May 4, 2003, was a Tunisian neurologist and neuropsychiatrist. Education After finishing his primary education in Kairouan, Ain drahem and Kelibia, Mongi Ben Hamida completed his secondary education at Sadiki College, where he got his baccalaureate degree in 1948. Later, he enrolled in medical studies at the Medical School of Paris (divided in 1970) where he specialized in neurology. His thesis on the dento-olivary pair in 1965 won him a Prix de Thèse (thesis prize) and quickly became an international clinical and neuropathological reference. Mongi Ben Hamida held a diploma of advanced studies in histology and cytology under the direction of Professor René Couteaux. Medical career During his stay in France, Mongi Ben Hamida worked as a Clinic Chief at the Salpêtrière hospital, in the unit of Professor Raymond Garcin, then in Professor Boudin's, a great master of French n ... [...More Info...] [...Related Items...] OR: [Wikipedia] [Google] [Baidu] |
Arabic
Arabic (, ' ; , ' or ) is a Semitic language spoken primarily across the Arab world.Semitic languages: an international handbook / edited by Stefan Weninger; in collaboration with Geoffrey Khan, Michael P. Streck, Janet C. E.Watson; Walter de Gruyter GmbH & Co. KG, Berlin/Boston, 2011. Having emerged in the 1st century, it is named after the Arab people; the term "Arab" was initially used to describe those living in the Arabian Peninsula, as perceived by geographers from ancient Greece. Since the 7th century, Arabic has been characterized by diglossia, with an opposition between a standard prestige language—i.e., Literary Arabic: Modern Standard Arabic (MSA) or Classical Arabic—and diverse vernacular varieties, which serve as mother tongues. Colloquial dialects vary significantly from MSA, impeding mutual intelligibility. MSA is only acquired through formal education and is not spoken natively. It is the language of literature, official documents, and formal writ ... [...More Info...] [...Related Items...] OR: [Wikipedia] [Google] [Baidu] |
Myopathy
In medicine, myopathy is a disease of the muscle in which the muscle fibers do not function properly. This results in muscular weakness. ''Myopathy'' means muscle disease ( Greek : myo- ''muscle'' + patheia '' -pathy'' : ''suffering''). This meaning implies that the primary defect is within the muscle, as opposed to the nerves ("neuropathies" or " neurogenic" disorders) or elsewhere (e.g., the brain). Muscle cramps, stiffness, and spasm can also be associated with myopathy. Capture myopathy can occur in wild or captive animals, such as deer and kangaroos, and leads to morbidity and mortality. It usually occurs as a result of stress and physical exertion during capture and restraint. Muscular disease can be classified as neuromuscular or musculoskeletal in nature. Some conditions, such as myositis, can be considered both neuromuscular and musculoskeletal. Signs and symptoms Common symptoms include muscle weakness, cramps, stiffness, and tetany. Systemic diseases Myo ... [...More Info...] [...Related Items...] OR: [Wikipedia] [Google] [Baidu] |
Neurodegeneration
A neurodegenerative disease is caused by the progressive loss of structure or function of neurons, in the process known as neurodegeneration. Such neuronal damage may ultimately involve cell death. Neurodegenerative diseases include amyotrophic lateral sclerosis, multiple sclerosis, Parkinson's disease, Alzheimer's disease, Huntington's disease, multiple system atrophy, and prion diseases. Neurodegeneration can be found in the brain at many different levels of neuronal circuitry, ranging from molecular to systemic. Because there is no known way to reverse the progressive degeneration of neurons, these diseases are considered to be incurable; however research has shown that the two major contributing factors to neurodegeneration are oxidative stress and inflammation. Biomedical research has revealed many similarities between these diseases at the subcellular level, including atypical protein assemblies (like proteinopathy) and induced cell death. These similarities suggest t ... [...More Info...] [...Related Items...] OR: [Wikipedia] [Google] [Baidu] |
Vitamin E Deficiency
Vitamin E deficiency in humans is a very rare condition, occurring as a consequence of abnormalities in dietary fat absorption or metabolism rather than from a diet low in vitamin E. Collectively the EARs, RDAs, AIs and ULs for vitamin E and other essential nutrients are referred to as Dietary Reference Intakes (DRIs). Vitamin E deficiency can cause nerve problems due to poor conduction of electrical impulses along nerves due to changes in nerve membrane structure and function. Signs and symptoms Signs of vitamin E deficiency include the following: * Neuromuscular problems – such as spinocerebellar ataxia and myopathies. * Neurological problems – may include dysarthria, absence of deep tendon reflexes, loss of the ability to sense vibration and detect where body parts are in three dimensional space, and positive Babinski sign. * Hemolytic anemia – due to oxidative damage to red blood cells * Retinopathy * Impairment of the immune response Causes Vitamin E deficiency is ... [...More Info...] [...Related Items...] OR: [Wikipedia] [Google] [Baidu] |
Friedreich's Ataxia
Friedreich's ataxia (FRDA or FA) is an autosomal-recessive genetic disease that causes difficulty walking, a loss of sensation in the arms and legs, and impaired speech that worsens over time. Symptoms generally start between 5 and 20 years of age. Many develop hypertrophic cardiomyopathy and require a mobility aid such as a cane, walker, or wheelchair in their teens. As the disease progresses, some affected people lose their sight and hearing. Other complications may include scoliosis and diabetes mellitus. The condition is caused by mutations in the ''FXN'' gene on chromosome 9, which makes a protein called frataxin. In FRDA, cells produce less frataxin. Degeneration of nerve tissue in the spinal cord causes the ataxia; particularly affected are the sensory neurons essential for directing muscle movement of the arms and legs through connections with the cerebellum. The spinal cord becomes thinner, and nerve cells lose some myelin sheath. No effective treatment ... [...More Info...] [...Related Items...] OR: [Wikipedia] [Google] [Baidu] |
Fayçal Hentati
Faisal, Faisel, Fayçal or Faysal ( ar, فيصل) is an Arabic given name. Faisal, Fayçal or Faysal may also refer to: People * King Faisal (other) ** Faisal I of Iraq and Syria (1885–1933), leader during the Arab Revolt ** Faisal II of Iraq (1935–1958), last King of the Kingdom of Iraq ** Faisal of Saudi Arabia (1906–1975), third King of Saudi Arabia * Faisal al-Duwaish (1882–1931), Arabian tribe sheik * Faisal Karami (born 1971), Lebanese politician * Faisal bin Abdullah Al Saud (born 1950), Saudi royal * Faisal bin Bandar Al Saud (born 1945), Saudi government official * Faisal bin Bandar Al Saud, Saudi royal and businessman * Faisal bin Khalid Al Saud (born 1973), Saudi government official * Faisal bin Mishaal Al Saud (born 1959), Saudi government official * Faisal bin Musaid Al Saud, Saudi royal * Faisal bin Sattam Al Saud (born 1970), Saudi ambassador to Italy * Faisal bin Turki Al Saud, Saudi royal * Faisal bin Turki I Al Saud (1920–1968), Saudi ... [...More Info...] [...Related Items...] OR: [Wikipedia] [Google] [Baidu] |
Charcot–Marie–Tooth Disease
Charcot–Marie–Tooth disease (CMT) is a hereditary motor and sensory neuropathy of the peripheral nervous system characterized by progressive loss of muscle tissue and touch sensation across various parts of the body. This disease is the most commonly inherited neurological disorder, affecting about one in 2,500 people. It is named after those who classically described it: the Frenchman Jean-Martin Charcot (1825–1893), his pupil Pierre Marie (1853–1940), and the Briton Howard Henry Tooth (1856–1925). There is no known cure. Care focuses on maintaining function. CMT was previously classified as a subtype of muscular dystrophy. Signs and symptoms Symptoms of CMT usually begin in early childhood or early adulthood but can begin later. Some people do not experience symptoms until their early 30s or 40s. Usually, the initial symptom is foot drop early in the course of the disease. This can also cause hammertoe, where the toes are always curled. Wasting of muscle tissue of ... [...More Info...] [...Related Items...] OR: [Wikipedia] [Google] [Baidu] |
Phenotype
In genetics, the phenotype () is the set of observable characteristics or traits of an organism. The term covers the organism's morphology or physical form and structure, its developmental processes, its biochemical and physiological properties, its behavior, and the products of behavior. An organism's phenotype results from two basic factors: the expression of an organism's genetic code, or its genotype, and the influence of environmental factors. Both factors may interact, further affecting phenotype. When two or more clearly different phenotypes exist in the same population of a species, the species is called polymorphic. A well-documented example of polymorphism is Labrador Retriever coloring; while the coat color depends on many genes, it is clearly seen in the environment as yellow, black, and brown. Richard Dawkins in 1978 and then again in his 1982 book '' The Extended Phenotype'' suggested that one can regard bird nests and other built structures such as ... [...More Info...] [...Related Items...] OR: [Wikipedia] [Google] [Baidu] |
Amyotrophic Lateral Sclerosis
Amyotrophic lateral sclerosis (ALS), also known as motor neuron disease (MND) or Lou Gehrig's disease, is a neurodegenerative disease that results in the progressive loss of motor neurons that control voluntary muscles. ALS is the most common type of motor neuron diseases. Early symptoms of ALS include stiff muscles, muscle twitches, and gradual increasing weakness and muscle wasting. ''Limb-onset ALS'' begins with weakness in the arms or legs, while ''bulbar-onset ALS'' begins with difficulty speaking or swallowing. Half of the people with ALS develop at least mild difficulties with thinking and behavior, and about 15% develop frontotemporal dementia. Most people experience pain. The affected muscles are responsible for chewing food, speaking, and walking. Motor neuron loss continues until the ability to eat, speak, move, and finally the ability to breathe is lost. ALS eventually causes paralysis and early death, usually from respiratory failure. Most cases of ALS ... [...More Info...] [...Related Items...] OR: [Wikipedia] [Google] [Baidu] |
Kamel Ben Othman
Kamel ( ar, كامل }) is a given name meaning ''perfect'' or ''the perfect one''. It may refer to: People with the given name Kamel * Abdullah Kamel Abdullah Kamel Al Kandari (born 1973), Kuwaiti extrajudicial prisoner of the United States * Hassan Kamel Al-Sabbah (1895–1935), Lebanese engineer * Kamel Ajlouni (born 1943), Jordanian endocrinologist * Kamel al-Budeiri (1882–1923), Palestinian politician * Kamel al-Kilani (born 1958), Iraqi politician * Kamel Al-Mousa (born 1982), Saudi Arabian football player * Kamel Asaad (1932–2010), Lebanese politician * Kamel Ayari, Tunisian wheelchair racer * Kamel Boughanem (born 1979), Moroccan-French football player * Kamel Chafni (born 1982), Moroccan football player * Kamel Ghilas (born 1984), Algerian football player * Kamel Hana Gegeo (died 1988), Iraqi murder victim * Kamel Habri (born 1976), Algerian football player * Kamel Kardjena (born 1981), Algerian Paralympic athlete * Kamel Lemoui, Algerian footballer * Kamel Maghur ... [...More Info...] [...Related Items...] OR: [Wikipedia] [Google] [Baidu] |
Duchenne Muscular Dystrophy
Duchenne muscular dystrophy (DMD) is a severe type of muscular dystrophy that primarily affects boys. Muscle weakness usually begins around the age of four, and worsens quickly. Muscle loss typically occurs first in the thighs and pelvis followed by the arms. This can result in trouble standing up. Most are unable to walk by the age of 12. Affected muscles may look larger due to increased fat content. Scoliosis is also common. Some may have intellectual disability. Females with a single copy of the defective gene may show mild symptoms. The disorder is X-linked recessive. About two thirds of cases are inherited from a person's mother, while one third of cases are due to a new mutation. It is caused by a mutation in the gene for the protein dystrophin. Dystrophin is important to maintain the muscle fiber's cell membrane. Genetic testing can often make the diagnosis at birth. Those affected also have a high level of creatine kinase in their blood. Although there is no known ... [...More Info...] [...Related Items...] OR: [Wikipedia] [Google] [Baidu] |
Medicine School Of Tunis
Medicine School of Tunis(arabic: ) (french: ) (FMT) is one of the four medical schools in Tunisia. It is a public institution that belongs to the Tunis El Manar University. History The school was founded in 1964 with only 59 students enrolled. The courses took place in the human and social sciences faculty of Tunis in that year. In 1965, they changed into the University Hospital Charles-Nicolle. Amor Chadli was the first dean of the school. Faculté de médecine de Tunis en 1964-1965.jpg, Medicine School of Tunis in 1964–65 Fac Med Tunis.JPG, Old building of the Medicine School of Tunis Debating club at FMT.jpg, Debating club at the medicine school of Tunis. Location The school is located in 15 Djebel Lakhdhar Street La Rabta 1007 in Tunis . Rue de la Faculté de médecine نهج كلية الطب.jpg, Medicine School of Tunis Street La cour de la faculté de medecine de tunis.JPG, Hall of the Medicine School of Tunis Main Hall of FMT.jpg, Main hall of the school Hal ... [...More Info...] [...Related Items...] OR: [Wikipedia] [Google] [Baidu] |
