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NM_005760

NM_001024806
NM_009882

RefSeq (protein)

NP_005751

NP_001019977

Location (UCSC) Chr 2: 37.2 – 37.23 Mb Chr 17: 78.92 – 78.94 Mb PubMed search [3] [4] Wikidata
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CCAAT/enhancer-binding protein zeta is a protein that in humans is encoded by the CEBPZ gene.[5][6][7]

Interactions

CCAAT/enhancer binding protein zeta has been shown to interact with:

See also

References

  1. ^ a b c GRCh38: Ensembl release 89: ENSG00000115816 - Ensembl, May 2017
  2. ^ a b c GRCm38: Ensembl release 89: ENSMUSG00000024081 - Ensembl, May 2017
  3. ^ "Human PubMed Reference:". 
  4. ^ "Mouse PubMed Reference:". 
  5. ^ Lum LS, Sultzman LA, Kaufman RJ, Linzer DI, Wu BJ (Jan 1991). "A cloned human CCAAT-box-binding factor stimulates transcription from the human hsp70 promoter". Mol Cell Biol. 10 (12): 6709–17. PMC 362949Freely accessible. PMID 2247079. 
  6. ^ a b Uramoto H, Izumi H, Nagatani G, Ohmori H, Nagasue N, Ise T, Yoshida T, Yasumoto K, Kohno K (Apr 2003). "Physical interaction of tumour suppressor p53/p73 with CCAAT-binding transcription factor 2 (CTF2) and differential regulation of human high-mobility group 1 (HMG1) gene expression". Biochem J. 371 (Pt 2): 301–10. doi:10.1042/BJ20021646. PMC 1223307Freely accessible. PMID 12534345. 
  7. ^ "Entrez Gene: CEBPZ CCAAT/enhancer binding protein zeta". 
  8. ^ Imbriano C, Bolognese F, Gurtner A, Piaggio G, Mantovani R (Jul 2001). "HSP-CBF is an NF-Y-dependent coactivator of the heat shock promoters CCAAT boxes". J. Biol. Chem. 276 (28): 26332–9. doi:10.1074/jbc.M101553200. PMID 11306579. 
  9. ^ Uramoto H, Izumi H, Nagatani G, Ohmori H, Nagasue N, Ise T, Yoshida T, Yasumoto K, Kohno K (Apr 2003). "Physical interaction of tumour suppressor p53/p73 with CCAAT-binding transcription factor 2 (CTF2) and differential regulation of human high-mobility group 1 (HMG1) gene expression". Biochem. J. 371 (Pt 2): 301–10. doi:10.1042/BJ20021646. PMC 1223307Freely accessible. PMID 12534345. 

Further reading

External links

This article incorporates text from the United States National Library of Medicine, which is in the public domain.